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MED], UV- B/UV- A2 source). Insoluble, partially degraded collagen in the dermis was quantified. Results are mean В± SEM (error bars) from 6 subjects. SS indicates solar simulator. Place holder to copy figure label and caption. Figure 5. A, Ultraviolet B/UV- A2 irradiation induction of matrix- degrading metalloproteinase- 1 (MMP- 1) and DNA damage are greater in light skin compared with dark skin. Human skin color value (L*) was determined by a color meter (Minolta CR2. Chroma Meter). L* is a measure of color value (lightness/darkness), and varies between 0 (dark) and 1. Subjects with skin L* greater than 6. UV- B/UV- A2 (average exposure, 6. J/cm. 2 total UV irradiance). Subjects with L* less than 5. J/cm. 2 total UV irradiance (equivalent to 4 times the minimal erythema dose for the light skinned group) and 2. J/cm. 2 total UV irradiance (equivalent to 8 times minimal erythema dose for the light skinned group). Skin samples were obtained 2. UV irradiation and analyzed for matrix MMP- 1 messenger RNA (m. RNA) levels by real- time reverse transcriptase polymerase chain reaction, and DNA thymine dimer photoproducts, by immunofluorescence. B, Thymine dimer immunofluorescence data are representative of 5 light skinned subjects exposed to twice the minimal erythema dose and 5 dark skin subjects exposed to 2. J/cm. 2 (equivalent to 8 times the minimal erythema dose for light skinned group). Place holder to copy figure label and caption. Figure 6. A, Ultraviolet A1 irradiation induction of matrix- degrading metalloproteinase- 1 (MMP- 1) and DNA damage are greater in light skin compared with dark skin. Human skin color value (L*) was determined as described in the legend to Figure 5. Subjects were exposed to the indicated doses of UV- A1, and skin samples were obtained 2. UV irradiation. Matrix metalloproteinase- 1 messenger RNA (m. 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